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Omezzol (Omeprazole)

Omezzol is indicated for the short term treatment of peptic ulcer disease in adults where most patients heal within four weeks. Patients with duodenal ulcer disease and H. pylori infection disease that is active for up to one year may benefit from combination therapy that includes Omezzol with a macrolide antibiotic. Studies show a reduction in recurrence of duodenal ulcers when H. Pylori is treated and a reduced rate of clarithromycin resistance with triple therapy. Resistance should be expected and susceptibility testing performed if patients fail treatment and treatment should be adjusted accordingly. Omezzol is also indicated for gastric ulcers in adults, and gastroesophageal reflux disease in adults and pediatric populations. Studies have shown the efficacy of Omezzol for short term treatment in erosive esophagitis. Omezzol is also indicated for healing erosive esophagitis in both adults and children. Conditions prone to hypersecretion such as Zollinger-Ellison syndrome, multiple endocrine adenomas, and systemic mastocytosis also respond to management with Omezzol treatment in adults.

Other names for this medication:
Omeprazole, Prilosec, Acimax, Acimed, Agastin, Agrixal, Alboz, Alsidol, Altosec, Anadir, Antra, Antramups, Aprazole, Asec, Aspra, Audazol, Biocid, Bioprazol, Brux, Buscogast, Candazol, Ceprandal, Cizole, Cosec, Criogel, Danlox, Desec, Domer, Dudencer, Elcontrol, Elgam, Elkostop, Emage, Emeproton, Emez, Emilok, Erbolin, Eselan, Exter, Ezol, Flusal, Fordex, Gasec, Gastec, Gastracid, Gastral, Gastrimut, Gastrium, Gastronorm, Gastroplex, Gastrotem, Gastrozol, Gastrozole, Getzome, Glaveral, Grizol, Helicid, Hycid, Inhibitron, Inhipump, Inpro, Ipproton, Klomeprax, Lanex, Lenar, Limnos, Locid, Locimez, Logastric, Lokev, Lokit, Lomac, Lomex, Lopraz, Lordin, Losamel, Losec, Losectil, Losepine, Loseprazol, Medoprazole, Mepral, Meprazol, Merofex, Metsec, Minisec, Miol, Miracid, Mopral, Nansen, Nocid, Nogacid, Norpramin, Norsec, Novek, Nozer, Nuclosina, Ocid, Ogal, Olark, Olexin, Olit, Omag, Omalcer, Omapren, Omapro, Omax, Omebeta, Omec, Omecap, Omecip, Omedar, Omel, Omep, Omepra, Omepradex, Omepral, Omeprazol, Omeprazolum, Omepren, Omeprol, Omepron, Omeral, Omeran, Omesec, Omestad, Ometab, Ometac, Omez, Omezol, Omezolan, Omezole, Omezyn, Omezzol, Omicap, Omig, Omisec, Omiz, Omizac, Omolin, Onic, Opaz, Opep, Oprazole, Oraz, Ortanol, Ozid, Parizac, Penrazol, Pepticum, Peptidin, Polprazol, Pratiprazol, Pravil, Prazidec, Prazol, Prazole, Prazolen, Prazolit, Presec, Prevas, Probitor, Procap, Procelac, Proceptin, Proclor, Prohibit, Prolok, Promisec, Protoloc, Protop, Prysma, Pumpitor, Regulacid, Risek, Sanamidol, Seclo, Sopral, Stomex, Tulzol, Ulceral, Ulcesep, Ulcid, Ulcosan, Ulcozol, Ulsen, Ultop, Ulzol, Xoprin, Zanprol, Zefxon, Zegerid, Zenpro, Zepral, Zolcer, Zollocid, Zoltum, Zomepral,

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Omezzol reduces the amount of acid your stomach makes. It's a widely used treatment for indigestion and heartburn and acid reflux. It's also taken to prevent and treat stomach ulcers.

Sometimes Omezzol is taken for a rare illness caused by a tumour in the pancreas or gut called Zollinger-Ellison syndrome.

Omezzol comes as capsules, tablets and as a liquid that you swallow (this is made to order).

All types of Omezzol are available on prescription. You can buy the lowest strength 10mg tablets and capsules from pharmacies.

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Soluble CD40 ligand as a predictor of coronary artery disease and long-term clinical outcomes in stable patients undergoing coronary angiography. Argatroban (administered as bolus or continuous infusion), UFH (bolus), and enoxaparin (bolus) significantly reduced catheter thrombus formation compared to untreated controls. The apparent increase in male reproductive tract disorders is briefly discussed from the perspective of the potential endocrine-disrupting effects of the numerous chemicals in the environment to which the developing male foetus can be exposed.

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Since NF-kappaB-mediated gene products, such as fibrinogen and PAI-1, are known to facilitate hypercoagulation, thrombosis and vascular events, we suggest that nilvadipine has a direct beneficial effect separate from its anti-hypertensive properties by inhibiting NF-kappaB-dependent gene expression and eventually inhibiting atherosclerosis. The aim of this study was to determine plasma CCK concentrations during fasting and in response to small-intestine nutrient infusion in critically ill patients. The expression of 45 genes was significantly changed in the early stage after spinal cord injury, in which 22 genes up-regulated and 23 genes down-regulated. We considered that these findings show narrowing of the MCA was due to cerebral arteritis.

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Dynamic study of hepatocellular pathological change and uptake rate of 125I-insulin during experimental hepatocarcinogenesis Thirty quinoxalines bearing a substituted phenoxy or hydroxybenzoylglutamate group on position 2, a carboethoxy or carboxy group on position 3 and a trifluoromethyl group on position 6 or 7 of the heterocycle were prepared in order to evaluate the in vitro anticancer activity. Role of copper in mitochondrial biogenesis via interaction with ATP synthase and cytochrome c oxidase. These results and their understanding should allow a different approach of biomechanical considerations, ie, a large-size square wire does not produce necessarily high forces. Forces appear to be transduced at the cell-ECM interface via re-arrangement of cytoskeletal elements, activation of stretch-induced changes in ion channels, cell contraction at adherens junctions, activation of cell membrane-associated secondary messenger pathways and through growth factor-like activities that influence cellular proliferation and protein synthesis.

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Apoptosis and cell-cycle analysis were performed by flow cytometry. In (A/J x C57BL/6J)F1 mouse lung tumors, the Lmna-rs1 gene was completely downregulated, whereas allele-specific downregulation of the C57BL/6J-derived allele was observed at the Las1 gene, suggesting the potential role of these genes in tumor suppression. Tympanosclerosis is a scarring process with a remarkable variability in its localization within the middle ear. Methods and Results: Four Polish patients with PTPS deficiency were screened for mutations in the PTS gene. The substances were considered to contribute to foam cell recruitment during the persistence of xanthoma lesions.

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